RCMI Coordinating Center (RCMI CC) Header Logo

Search Result Details

This page shows the details of why an item matched the keywords from your search.
One or more keywords matched the following items that are connected to Zhang, John
Item TypeName
Concept Protein Kinase C
Concept Mitogen-Activated Protein Kinase 1
Concept Mitogen-Activated Protein Kinase Kinases
Concept Protein Kinase Inhibitors
Concept Mitogen-Activated Protein Kinase 3
Concept Mitogen-Activated Protein Kinase 7
Concept Proto-Oncogene Proteins c-akt
Concept Protein Kinase C-epsilon
Academic Article Administration of rCTRP9 Attenuates Neuronal Apoptosis Through AdipoR1/PI3K/Akt Signaling Pathway after ICH in Mice.
Academic Article Critical role of EphA4 in early brain injury after subarachnoid hemorrhage in rat.
Academic Article CRMP-2 is involved in axon growth inhibition induced by RGMa in vitro and in vivo.
Academic Article Adiponectin attenuates neuronal apoptosis induced by hypoxia-ischemia via the activation of AdipoR1/APPL1/LKB1/AMPK pathway in neonatal rats.
Academic Article Remote Limb Ischemic Preconditioning Attenuates Cerebrovascular Depression During Sinusoidal Galvanic Vestibular Stimulation via a1-Adrenoceptor-Protein Kinase Ce-Endothelial NO Synthase Pathway in Rats.
Academic Article Activation of TGR5 with INT-777 attenuates oxidative stress and neuronal apoptosis via cAMP/PKCe/ALDH2 pathway after subarachnoid hemorrhage in rats.
Academic Article Inhibitory effect with antisense mitogen-activated protein kinase oligodeoxynucleotide against cerebral vasospasm in rats.
Academic Article Dimethyl fumarate confers neuroprotection by casein kinase 2 phosphorylation of Nrf2 in murine intracerebral hemorrhage.
Academic Article Contribution of Src tyrosine kinase to cerebral vasospasm after subarachnoid hemorrhage.
Academic Article PKGIa inhibits the proliferation of cerebral arterial smooth muscle cell induced by oxyhemoglobin after subarachnoid hemorrhage.
Academic Article Aggf1 attenuates neuroinflammation and BBB disruption via PI3K/Akt/NF-?B pathway after subarachnoid hemorrhage in rats.
Academic Article Role of PDGF-D and PDGFR-? in neuroinflammation in experimental ICH mice model.
Academic Article Simvastatin attenuation of cerebral vasospasm after subarachnoid hemorrhage in rats via increased phosphorylation of Akt and endothelial nitric oxide synthase.
Academic Article Curcumin inhibits microglia inflammation and confers neuroprotection in intracerebral hemorrhage.
Academic Article Rh-relaxin-2 attenuates degranulation of mast cells by inhibiting NF-?B through PI3K-AKT/TNFAIP3 pathway in an experimental germinal matrix hemorrhage rat model.
Academic Article Kisspeptin-54 attenuates oxidative stress and neuronal apoptosis in early brain injury after subarachnoid hemorrhage in rats via GPR54/ARRB2/AKT/GSK3? signaling pathway.
Academic Article Intranasal administration of recombinant Netrin-1 attenuates neuronal apoptosis by activating DCC/APPL-1/AKT signaling pathway after subarachnoid hemorrhage in rats.
Academic Article Tozasertib attenuates neuronal apoptosis via DLK/JIP3/MA2K7/JNK pathway in early brain injury after SAH in rats.
Academic Article FGF-2 Attenuates Neuronal Apoptosis via FGFR3/PI3k/Akt Signaling Pathway After Subarachnoid Hemorrhage.
Academic Article Intranasal administration of recombinant prosaposin attenuates neuronal apoptosis through GPR37/PI3K/Akt/ASK1 pathway in MCAO rats.
Academic Article Neurotrophin-3 provides neuroprotection via TrkC receptor dependent pErk5 activation in a rat surgical brain injury model.
Academic Article P2X7 receptor antagonism inhibits p38 mitogen-activated protein kinase activation and ameliorates neuronal apoptosis after subarachnoid hemorrhage in rats.
Search Criteria
  • Proto Oncogene Proteins c akt
RCMI CC is supported by the National Institute on Minority Health and Health Disparities, National Institutes of Health (NIH), through Grant Number U24MD015970. The contents of this site are solely the responsibility of the authors and do not necessarily represent the official views of the NIH

For technical support please contact support